Disease Info Card

Transfusion Associated Circulatory Overload

Information about Transfusion Associated Circulatory Overload: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Transfusion Associated Circulatory Overload

Most recent studies have shown that Transfusion Associated Circulatory Overload shares some biological mechanisms with acute-lung-injury, acute-pulmonary-edema, allergy, anaphylaxis, anemia, cardiovascular-diseases, corneal-diseases, dyspnea, edema, fluid-overload, heart-failure, lung-injury, obesity, pulmonary-edema, respiratory-distress-syndrome-adult, respiratory-failure, transfusion-reaction, transfusion-related-acute-lung-injury, transfusion-related-acute-lung-injury-(trali), tuberculosis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Transfusion Associated Circulatory Overload, and have been seen in publications frequently: Anaphylaxis, Immune Response, Innate Immune Response, Localization, Methylation, Opsonization, Pathogenesis, Phagocytosis, Phagosome Maturation, Phagosome-lysosome Fusion, Reverse Transcription, Secretion, Transport, Virulence

Quite a number of genes have been found to play important roles in Transfusion Associated Circulatory Overload, such as ATR, BBS9, CORO1A, EEA1, GNAI1, GUSB, KIT, LAMP1, MS4A1, NDUFA2, NPPB, NR1H4, SLC4A7, SPANXB1, TLR2, TLR4, VDR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.