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Disease Info Card
Splenic Marginal Zone B-cell Lymphoma
Information about Splenic Marginal Zone B-cell Lymphoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Splenic Marginal Zone B-cell Lymphoma
Most recent studies have shown that Splenic Marginal Zone B-cell Lymphoma shares some biological mechanisms with b-cell-lymphomas, chronic-lymphocytic-leukemia, diffuse-large-b-cell-lymphoma, hairy-cell-leukemia, leukemia, lymphocytosis, lymphoid-leukemia, lymphoma, lymphoma-follicular, lymphoma-non-hodgkin, lymphoproliferative-disorders, malignant-lymphoma-lymphocytic-intermediate-differentiation-diffuse, malignant-neoplasms, malignant-paraganglionic-neoplasm, marginal-zone-b-cell-lymphoma, mucosa-associated-lymphoid-tissue-lymphoma, neoplasms, prolymphocytic-leukemia, splenic-lymphoma, splenic-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Splenic Marginal Zone B-cell Lymphoma, and have been seen in publications frequently: Cell Adhesion, Cell Cycle, Cell Development, Cell Differentiation, Cell Growth, Cell Proliferation, Coagulation, Dna Repair, Immune Response, Interphase, Localization, Lymphocyte Proliferation, Metaphase, Methylation, Mitosis, Oncogenesis, Pathogenesis, Plasma Cell Differentiation, Programmed Cell Death, Transport
Quite a number of genes have been found to play important roles in Splenic Marginal Zone B-cell Lymphoma, such as BCL2, BCL6, CCND1, CD19, CD22, CD5, CLEC4D, CTLA4, FCER2, FH, HLA-DQA1, IL2RA, ISG20, ITGAX, KRT20, MME, MS4A1, NOD2, TP53. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.