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Disease Info Card
Mucosa-associated Lymphoid Tissue Lymphoma
Information about Mucosa-associated Lymphoid Tissue Lymphoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Mucosa-associated Lymphoid Tissue Lymphoma
Most recent studies have shown that Mucosa-associated Lymphoid Tissue Lymphoma shares some biological mechanisms with adenocarcinoma, b-cell-lymphomas, carcinoma, diffuse-large-b-cell-lymphoma, gastric-lymphoma, gastric-mucosa-associated-lymphoid-tissue-lymphoma, gastritis, helicobacter-infections, helicobacter-pylori-infection, lymphoma, lymphoma-follicular, lymphoma-non-hodgkin, malignant-neoplasm-of-stomach, malignant-neoplasms, malignant-paraganglionic-neoplasm, marginal-zone-b-cell-lymphoma, neoplasms, peptic-ulcer, stomach-neoplasms, ulcer.
Among the many pathways, these few ones have gauged particular interests from scientists studying Mucosa-associated Lymphoid Tissue Lymphoma, and have been seen in publications frequently: Acid Secretion, B Cell Proliferation, Cell Activation, Cell Adhesion, Cell Cycle, Cell Death, Cell Differentiation, Cell Proliferation, Immune Response, Inflammatory Response, Interphase, Localization, Methylation, Oncogenesis, Pathogenesis, Plasma Cell Differentiation, Reverse Transcription, Secretion, Transport, Virulence
Quite a number of genes have been found to play important roles in Mucosa-associated Lymphoid Tissue Lymphoma, such as BCL10, BCL2, BCL6, BIRC3, CCND1, CD5, CHP1, CTLA4, DLC1, DYNC1H1, HLA-DQA1, HP, KRT20, MALT1, MME, MS4A1, NFKB1, NOD2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.