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Disease Info Card
Spermatogenesis Arrest
Information about Spermatogenesis Arrest: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Spermatogenesis Arrest
Most recent studies have shown that Spermatogenesis Arrest shares some biological mechanisms with abnormal-degeneration, albinism, atrophy, atrophy-of-testis, autoimmune-diseases, autoimmune-reaction, azoospermia, congenital-absence-of-germinal-epithelium-of-testes, cryptorchidism, growth-arrest, hyperplasia, infertility, male-infertility, malignant-neoplasms, neoplasms, oligospermia, orchitis, sterility, testicular-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Spermatogenesis Arrest, and have been seen in publications frequently: Cell Development, Cell Differentiation, Cell Maturation, Fertilization, Germ Cell Development, Hypersensitivity, Localization, Mating, Meiosis, Meiosis I, Metaphase, Pachytene, Pathogenesis, Prophase, Secretion, Sperm Motility, Spermatogenesis, Testosterone Secretion, Translation, Zygotene
Quite a number of genes have been found to play important roles in Spermatogenesis Arrest, such as AKR1B1, AR, AREG, BRD2, C1QBP, CCNA1, CREM, DAZ1, DDX25, ERMAP, EXOSC10, FDXR, GJA1, GNRH1, PLOD1, POMC, PRL, SYCP3, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.