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Disease Info Card
Congenital Absence Of Germinal Epithelium Of Testes
Information about Congenital Absence Of Germinal Epithelium Of Testes: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Congenital Absence Of Germinal Epithelium Of Testes
Most recent studies have shown that Congenital Absence Of Germinal Epithelium Of Testes shares some biological mechanisms with aplasia-nos, atrophy, azoospermia, cryptorchidism, cytogenetic-abnormality, growth-arrest, hypogonadism, infertility, klinefelter-syndrome, male-infertility, malignant-neoplasms, neoplasms, oligospermia, partial-deletion-of-y, sex-chromosome-aberrations, spermatogenesis-arrest, spindle-cell-oncocytoma-of-the-adenohypophysis, steroid-sulfatase-deficiency-disease, testicular-diseases, testicular-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Congenital Absence Of Germinal Epithelium Of Testes, and have been seen in publications frequently: Cell Cycle, Cell Development, Cell Differentiation, Cell Maturation, Cell Proliferation, Fertilization, Germ Cell Development, Localization, Meiosis, Metaphase, Pachytene, Pathogenesis, Prophase, Reverse Transcription, Secretion, Sex Determination, Sperm Motility, Spermatogenesis, Translation, Transport
Quite a number of genes have been found to play important roles in Congenital Absence Of Germinal Epithelium Of Testes, such as AR, BPY2, BRD2, CD19, CDY1, CFTR, DAZ1, DAZ2, DAZL, DDX3Y, GJA1, KDM5D, PLOD1, PRL, PRM2, RBMY1A1, SRY, STS, USP9Y. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.