This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Semantic Dementia
Information about Semantic Dementia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Semantic Dementia
Most recent studies have shown that Semantic Dementia shares some biological mechanisms with alzheimers-disease, anomia, aphasia, aphasia-broca, atrophy, cognition-disorders, dementia, frontotemporal-dementia, frontotemporal-lobar-degeneration, language-disorders, malnutrition, memory-disorders, motor-neuron-disease, neurodegenerative-disorders, non-fluent-aphasia, pick-disease-of-the-brain, primary-progressive-aphasia-(disorder), primary-progressive-nonfluent-aphasia, progressive-aphasia, semantic-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Semantic Dementia, and have been seen in publications frequently: Aging, Associative Learning, Cognition, Eating Behavior, Intraflagellar Transport, Localization, Locomotion, Long-term Memory, Muscle Atrophy, Pathogenesis, Response To Pain, Segmentation, Short-term Memory, Social Behavior, Translation, Transport
Quite a number of genes have been found to play important roles in Semantic Dementia, such as AMY2A, BLOC1S6, CBS, EPB42, FUS, GRN, IGFALS, LPA, MAPT, PRH1, RANGAP1, REG1A, SEMA3A, SLC17A5, SOD1, STXBP3, TARDBP, VCP. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.