Disease Info Card

Seborrheic Keratosis

Information about Seborrheic Keratosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Seborrheic Keratosis

Most recent studies have shown that Seborrheic Keratosis shares some biological mechanisms with actinic-keratosis, basal-cell-carcinoma, bowens-disease, carcinoma, common-wart, dermatologic-disorders, keratosis, lentigo, leser-trelat-sign, malignant-neoplasm-of-skin, malignant-neoplasms, malignant-paraganglionic-neoplasm, malignant-squamous-cell-neoplasm, melanocytic-nevus, melanoma, neoplasms, papilloma, psoriasis, seborrheic-dermatitis, skin-neoplasms.

Among the many pathways, these few ones have gauged particular interests from scientists studying Seborrheic Keratosis, and have been seen in publications frequently: Aging, Cell Cycle, Cell Differentiation, Cell Growth, Cell Proliferation, Exocytosis, Immune Response, Keratinization, Keratinocyte Differentiation, Keratinocyte Proliferation, Localization, Oncogene-induced Senescence, Pathogenesis, Pigmentation, Regulation Of Cell Growth, Secretion, Segmentation, Senescence, Swimming, Wound Healing

Quite a number of genes have been found to play important roles in Seborrheic Keratosis, such as ADK, BCL2, C2, CDKN1A, CDKN2A, CKAP4, DPP4, EGF, EGFR, FGFR3, HRAS, IVL, PCNA, PIK3CA, POMC, SERPINB3, TNF, TP53, TP63. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.