Disease Info Card

Pseudomembranous Colitis

Information about Pseudomembranous Colitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Pseudomembranous Colitis

Most recent studies have shown that Pseudomembranous Colitis shares some biological mechanisms with abdominal-pain, antibiotic-associated-diarrhea, antibiotic-enterocolitis, bacterial-infections, clostridium-difficile-infection, clostridium-infections, colitis, cross-infection, diarrhea, enterocolitis-pseudomembranous, infective-disorder, inflammation, intestinal-diseases, malignant-neoplasms, megacolon, megacolon-toxic, pain, pathological-dilatation, ulcer, ulcerative-colitis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Pseudomembranous Colitis, and have been seen in publications frequently: Acute Inflammatory Response, Anaphylaxis, Cell Death, Cell Proliferation, Coagulation, Dehiscence, Endocytosis, Excretion, Fermentation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Pathogenesis, Receptor-mediated Endocytosis, Response To Toxin, Secretion, Sporulation, Transport, Virulence

Quite a number of genes have been found to play important roles in Pseudomembranous Colitis, such as AKT1, ALB, ATP6V0A1, AZI2, CDC42, GNAI1, NAA25, NAP1L1, NAPSA, NCKAP1, RFC1, RFC2, RFC4, RHO, RHOA, RHOD, TAB3, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.