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- Table of Contents
Information about Clostridium Difficile Infection: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Clostridium Difficile Infection shares some biological mechanisms with antibiotic-associated-diarrhea, clostridium-infections, colitis, communicable-diseases, community-acquired-infections, cross-infection, diarrhea, enterocolitis-pseudomembranous, equine-infectious-anemia, infective-disorder, inflammation, inflammatory-bowel-diseases, intestinal-diseases, malignant-neoplasms, nosocomial-infection, pneumonia, pseudomembranous-colitis, recurrence-(disease-attribute), ulcer, ulcerative-colitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Clostridium Difficile Infection, and have been seen in publications frequently: Adaptive Immune Response, Aging, Cell Death, Coagulation, Drug Resistance, Excretion, Fermentation, Germination, Immune Response, Inflammatory Response, Pathogenesis, Phagocytosis, Platelet Activation, Platelet Aggregation, Reflex, Secretion, Spore Germination, Sporulation, Transposition, Virulence
Quite a number of genes have been found to play important roles in Clostridium Difficile Infection, such as ACAD8, ALB, AZI2, CACNA1A, GLUD1, GNAI1, H6PD, MTFMT, NAA25, NAP1L1, NAPSA, NCKAP1, NHS, SLC9A6, TAB3, TOP2A, TOX, UGDH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.