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Disease Info Card
Polyostotic Fibrous Dysplasia
Information about Polyostotic Fibrous Dysplasia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Polyostotic Fibrous Dysplasia
Most recent studies have shown that Polyostotic Fibrous Dysplasia shares some biological mechanisms with acromegaly, adenoma, bone-diseases, bone-neoplasms, dysplasia, endocrine-system-diseases, fibrous-dysplasia, fibrous-dysplasia-monostotic, fracture, hyperthyroidism, mccune-albright-syndrome, neoplasms, osteitis-fibrosa-cystica, osteitis-fibrosa-disseminata, pain, pituitary-diseases, pituitary-neoplasms, precocious-puberty, pseudohypoparathyroidism.
Among the many pathways, these few ones have gauged particular interests from scientists studying Polyostotic Fibrous Dysplasia, and have been seen in publications frequently: Bone Maturation, Bone Resorption, Cell Activation, Cell Proliferation, Estrogen Secretion, Excretion, Gonadotropin Secretion, Growth Hormone Secretion, Hormone Secretion, Localization, Menarche, Methylation, Ossification, Ovulation, Pathogenesis, Pigmentation, Secretion, Spermatogenesis, Thelarche, Transport
Quite a number of genes have been found to play important roles in Polyostotic Fibrous Dysplasia, such as APC, ASAH1, BRD2, CYP19A1, FGF23, GGH, GH1, GLUL, GNAS, IGF1, MAS1, PLOD1, PRL, PTH, PTRH1, SOX3, SST, TRH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.