Disease Info Card

Mccune-albright Syndrome

Information about Mccune-albright Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Mccune-albright Syndrome

Most recent studies have shown that Mccune-albright Syndrome shares some biological mechanisms with acromegaly, adenoma, cushing-syndrome, dysplasia, endocrine-system-diseases, fibrous-dysplasia, fracture, hyperplasia, hyperthyroidism, neoplasms, osteitis-fibrosa-cystica, osteitis-fibrosa-disseminata, ovarian-cysts, pain, pigmentation-disorders, pituitary-adenoma, pituitary-diseases, pituitary-neoplasms, polyostotic-fibrous-dysplasia, precocious-puberty.

Among the many pathways, these few ones have gauged particular interests from scientists studying Mccune-albright Syndrome, and have been seen in publications frequently: Bone Maturation, Bone Resorption, Cell Activation, Cell Cycle, Cell Proliferation, Estrogen Secretion, Excretion, Growth Hormone Secretion, Hormone Secretion, Localization, Menarche, Menstruation, Oncogenesis, Ovulation, Pathogenesis, Pigmentation, Secretion, Spermatogenesis, Thelarche, Transport

Quite a number of genes have been found to play important roles in Mccune-albright Syndrome, such as APC, BRD2, CYP19A1, FGF23, GGH, GH1, GLUL, GNAS, GNRH1, IGF1, MAS1, PBX1, PLOD1, POMC, PRL, PTH, PTRH1, SST, TRH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.