Disease Info Card

Fibrous Dysplasia

Information about Fibrous Dysplasia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Fibrous Dysplasia

Most recent studies have shown that Fibrous Dysplasia shares some biological mechanisms with bone-cysts, bone-cysts-aneurysmal, bone-diseases, bone-neoplasms, dysplasia, fibroma, fibrous-dysplasia-monostotic, fracture, malignant-paraganglionic-neoplasm, mccune-albright-syndrome, neoplasms, ossifying-fibroma, osteitis-fibrosa-cystica, osteitis-fibrosa-disseminata, osteoma, osteosarcoma, pain, polyostotic-fibrous-dysplasia, precocious-puberty, sarcoma.

Among the many pathways, these few ones have gauged particular interests from scientists studying Fibrous Dysplasia, and have been seen in publications frequently: Bone Development, Bone Maturation, Bone Mineralization, Bone Remodeling, Bone Resorption, Cell Proliferation, Dehiscence, Enucleation, Excretion, Localization, Menarche, Ossification, Osteoblast Differentiation, Pathogenesis, Pigmentation, Regeneration, Secretion, Transport, Wound Healing

Quite a number of genes have been found to play important roles in Fibrous Dysplasia, such as APC, BGLAP, BRD2, CALCA, FGF23, GGH, GH1, GLUL, GNAS, IGF1, IL6, MAS1, PLOD1, PRL, PTH, SST, TRH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.