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- Table of Contents
Information about Poikiloderma Of Kindler: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Poikiloderma Of Kindler shares some biological mechanisms with atrophic-condition-of-skin, atrophy, bulla, carcinoma, cicatrix, dermatologic-disorders, epidermolysis-bullosa, epidermolysis-bullosa-dystrophica, inflammation, malignant-neoplasms, malignant-squamous-cell-neoplasm, periodontal-diseases, photosensitivity-disorders, rothmund-thomson-syndrome, skin-diseases-genetic, skin-diseases-vesiculobullous, stenosis, tissue-adhesions.
Among the many pathways, these few ones have gauged particular interests from scientists studying Poikiloderma Of Kindler, and have been seen in publications frequently: Actin Cytoskeleton Organization, Aging, Cell Adhesion, Cell Migration, Cell Motility, Cell Proliferation, Cell-matrix Adhesion, Cytokine Secretion, Cytoskeleton Organization, Dna Repair, Enucleation, Inflammatory Response, Integrin Activation, Keratinocyte Migration, Localization, Pathogenesis, Pigmentation, Regeneration, Translation, Wound Healing
Quite a number of genes have been found to play important roles in Poikiloderma Of Kindler, such as CDH1, COL18A1, COL7A1, DST, EZR, FBLIM1, FERMT1, FERMT2, FERMT3, ILK, ITGB1, KRT14, KRT5, MSN, OXSM, PDLIM5, PLEK, RDX. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.