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Disease Info Card
Myocardial Reperfusion Injury
Information about Myocardial Reperfusion Injury: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Myocardial Reperfusion Injury
Most recent studies have shown that Myocardial Reperfusion Injury shares some biological mechanisms with acute-myocardial-infarction, arterial-occlusion, cardiac-arrhythmia, cardiac-fibrillation, coronary-heart-disease, coronary-occlusion, heart-diseases, heart-failure, hypoxia, infarction, inflammation, ischemia, myocardial-infarction, myocardial-ischemia, myocardial-stunning, reperfusion-arrhythmia, reperfusion-injury, ventricular-fibrillation.
Among the many pathways, these few ones have gauged particular interests from scientists studying Myocardial Reperfusion Injury, and have been seen in publications frequently: Aging, Angiogenesis, Autophagy, Cell Adhesion, Cell Death, Coagulation, Complement Activation, Electron Transport, Glycolysis, Inflammatory Response, Localization, Neutrophil Activation, Oxidative Phosphorylation, Pathogenesis, Platelet Aggregation, Response To Ischemia, Translation, Transport, Vasoconstriction, Vasodilation
Quite a number of genes have been found to play important roles in Myocardial Reperfusion Injury, such as AKT1, BCL2, CASP3, CAT, CHKA, CHKB, DLD, IL6, ITGB2, LAD1, MAPK1, MAPK3, MPO, NOS2, NOS3, PRRT2, SOD1, TNF, XDH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.