Disease Info Card

Mild Cognitive Disorder

Information about Mild Cognitive Disorder: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Mild Cognitive Disorder

Most recent studies have shown that Mild Cognitive Disorder shares some biological mechanisms with alzheimers-disease, amnesia, atrophy, carcinoma, cognition-disorders, dementia, dementia-vascular, depressive-disorder, impaired-cognition, impairment-(finding), malignant-neoplasm-of-thyroid, malignant-neoplasms, malnutrition, memory-disorders, memory-impairment, neoplasm-metastasis, neoplasms, neurodegenerative-disorders, parkinson-disease, thyroid-neoplasm.

Among the many pathways, these few ones have gauged particular interests from scientists studying Mild Cognitive Disorder, and have been seen in publications frequently: Aging, Associative Learning, Cell Death, Cognition, Excretion, Gastric Emptying, Hyperphosphorylation, Immune Response, Innervation, Localization, Long-term Memory, Menopause, Neurogenesis, Pathogenesis, Secretion, Segmentation, Sensitization, Short-term Memory, Translation, Transport

Quite a number of genes have been found to play important roles in Mild Cognitive Disorder, such as ACHE, AKR1C4, ALB, APOE, APP, BCHE, BDNF, CHN1, CSF2, FANCA, FXN, LAMC2, MAPT, NDUFB6, PSEN1, RUNX1T1, SLC6A3, SMUG1, TG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.