Leiomyoma, Multiple Cutaneous

Overview of Leiomyoma, Multiple Cutaneous

Most recent studies have shown that Leiomyoma, Multiple Cutaneous shares some biological mechanisms with benign-neoplasm, carcinoma, cutaneous-leiomyoma, dermatologic-disorders, fibroid-tumor, hereditary-diseases, hereditary-leiomyomatosis-and-renal-cell-cancer, kidney-neoplasm, leiomyomatosis, malignant-neoplasm-of-kidney, malignant-neoplasms, myoma, neoplasms, neoplasms-multiple-primary, neoplastic-syndromes-hereditary, pathologic-neovascularization, renal-cell-carcinoma, skin-neoplasms, uterine-fibroids, uterine-neoplasms.

Among the many pathways, these few ones have gauged particular interests from scientists studying Leiomyoma, Multiple Cutaneous, and have been seen in publications frequently: Angiogenesis, Glucose Transport, Glycolysis, Methylation, Mismatch Repair, Response To Cold, Transport

Quite a number of genes have been found to play important roles in Leiomyoma, Multiple Cutaneous, such as CFH, CHML, EXO1, FH, FHIT, HIF1A, KMO, LDLR, MET, MLH1, MSH2, OPN3, RASSF1, RGS7, SETD2, TWSG1, VHL. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Leiomyoma, Multiple Cutaneous Related Genes

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Pathways Related to Leiomyoma, Multiple Cutaneous

This information is being compiled and will come in a future update

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