This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Hyper Igd Syndrome
Information about Hyper Igd Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hyper Igd Syndrome
Most recent studies have shown that Hyper Igd Syndrome shares some biological mechanisms with amyloidosis, arthralgia, arthritis, brucellosis, chronic-infantile-neurological-cutaneous-and-articular-syndrome, deficiency-of-mevalonate-kinase, exanthema, familial-mediterranean-fever, hereditary-autoinflammatory-diseases, hypergammaglobulinemia, inflammation, mevalonic-aciduria, muckle-wells-syndrome, pain, relapsing-fever, tnf-receptor-associated-periodic-fever-syndrome-(traps), urticaria.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hyper Igd Syndrome, and have been seen in publications frequently: Acute Inflammatory Response, Acute-phase Response, B Cell Activation, Cell Activation, Cell Development, Cytokine Production, Cytokine Secretion, Excretion, Fucosylation, Glycosylation, Immune Response, Inflammatory Response, Innate Immune Response, Localization, Macrophage Activation, Pathogenesis, Regulation Of Cytokine Production, Reverse Transcription, Secretion, T Cell Activation
Quite a number of genes have been found to play important roles in Hyper Igd Syndrome, such as ATP7A, CRP, CSRP1, CTLA4, HLA-DQA1, HMGCR, IL1B, IL1RN, IL6, MDK, MEFV, MVK, NLRP3, NOD2, PSTPIP1, TEAD1, TNF, TNFRSF1A. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.