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Disease Info Card
Chronic Infantile Neurological, Cutaneous, And Articular Syndrome
Information about Chronic Infantile Neurological, Cutaneous, And Articular Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Chronic Infantile Neurological, Cutaneous, And Articular Syndrome
Most recent studies have shown that Chronic Infantile Neurological, Cutaneous, And Articular Syndrome shares some biological mechanisms with arthritis, arthritis-juvenile-rheumatoid, arthropathy, autoimmune-diseases, brucellosis, cns-disorder, cryopyrin-associated-periodic-syndromes, exanthema, familial-cold-urticaria, familial-mediterranean-fever, hereditary-autoinflammatory-diseases, hyper-igd-syndrome, inflammation, inflammatory-disorder, meningitis, muckle-wells-syndrome, nervousness, urticaria, urticaria-due-to-cold.
Among the many pathways, these few ones have gauged particular interests from scientists studying Chronic Infantile Neurological, Cutaneous, And Articular Syndrome, and have been seen in publications frequently: Acute-phase Response, Cell Proliferation, Central Nervous System Development, Chemotaxis, Cytokine Secretion, Endochondral Bone Growth, Granulocyte Activation, Immune Response, Inflammatory Response, Innate Immune Response, Interleukin-1 Production, Neutrophil Chemotaxis, Ossification, Pathogenesis, Phagocytosis, Regulation Of Interleukin-1 Production, Response To Lipopolysaccharide, Secretion, System Development, Translation
Quite a number of genes have been found to play important roles in Chronic Infantile Neurological, Cutaneous, And Articular Syndrome, such as CADPS, CAPS, CASP1, CRP, IL10, IL18, IL1B, IL1RN, MEFV, MVK, NFKB1, NLRP3, NOD2, PYCARD, S100A9, STS, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.