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Information about Deficiency Of Mevalonate Kinase: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Deficiency Of Mevalonate Kinase shares some biological mechanisms with abdominal-pain, arthritis, autoimmune-diseases, brucellosis, cryopyrin-associated-periodic-syndromes, exanthema, familial-mediterranean-fever, hereditary-autoinflammatory-diseases, hyper-igd-syndrome, hypergammaglobulinemia, inborn-errors-of-metabolism, inflammation, mevalonic-aciduria, pain, relapsing-fever, tnf-receptor-associated-periodic-fever-syndrome-(traps).
Among the many pathways, these few ones have gauged particular interests from scientists studying Deficiency Of Mevalonate Kinase, and have been seen in publications frequently: Acute Inflammatory Response, Acute-phase Response, Cell Activation, Cell Death, Cytokine Production, Cytokine Secretion, Excretion, Glycosylation, Immune Response, Inflammatory Response, Innate Immune Response, Interleukin-1 Beta Secretion, Pathogenesis, Programmed Cell Death, Protein Glycosylation, Regulation Of Cytokine Production, Response To Lipid, Secretion, T Cell Activation, Translation
Quite a number of genes have been found to play important roles in Deficiency Of Mevalonate Kinase, such as ATP7A, CADPS, CAPS, CASP1, CASP3, CRP, HMGCR, IL1B, IL1RN, IL4, MDK, MEFV, MVK, NLRP3, PSTPIP1, TNF, TNFRSF1A. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.