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- Table of Contents
Information about Muckle-wells Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Muckle-wells Syndrome shares some biological mechanisms with amyloidosis, arthralgia, arthritis, autoimmune-diseases, brucellosis, chronic-infantile-neurological-cutaneous-and-articular-syndrome, complete-hearing-loss, cryopyrin-associated-periodic-syndromes, exanthema, familial-cold-urticaria, familial-mediterranean-fever, hereditary-autoinflammatory-diseases, inflammation, inflammatory-disorder, kidney-diseases, sensorineural-hearing-loss-(disorder), urticaria, urticaria-due-to-cold.
Among the many pathways, these few ones have gauged particular interests from scientists studying Muckle-wells Syndrome, and have been seen in publications frequently: Acute-phase Response, Anaphylaxis, Cell Death, Cell Differentiation, Chemotaxis, Cytokine Secretion, Fertilization, Granulocyte Activation, Hypersensitivity, Immune Response, Inflammatory Response, Innate Immune Response, Neutrophil Activation, Neutrophil Chemotaxis, Pathogenesis, Protein Oligomerization, Reflex, Response To Lipopolysaccharide, Secretion, Translation
Quite a number of genes have been found to play important roles in Muckle-wells Syndrome, such as CADPS, CAPS, CASP1, CRP, IL18, IL1B, IL1RN, IL6, LYZ, MEFV, MVK, NLRP3, NOD2, PYCARD, STS, TEAD1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.