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Disease Info Card
Generalized Vitiligo
Information about Generalized Vitiligo: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Generalized Vitiligo
Most recent studies have shown that Generalized Vitiligo shares some biological mechanisms with arthritis, autoimmune-diseases, autoimmune-reaction, autoimmunity, bulla, dermatologic-disorders, diabetes-mellitus, diabetes-mellitus-insulin-dependent, hypopigmentation-disorder, koebner-phenomenon, localized-vitiligo, melanocytic-nevus, melanoma, overlying-behavior, psoriasis, rheumatoid-arthritis, thyroid-diseases, visual-halo.
Among the many pathways, these few ones have gauged particular interests from scientists studying Generalized Vitiligo, and have been seen in publications frequently: Cell Death, Cell Differentiation, Cell Growth, Cell Proliferation, Complement Activation, Habituation, Hypersensitivity, Immune Response, Innate Immune Response, Localization, Melanocyte Differentiation, Melanocyte Proliferation, Pathogenesis, Photoprotection, Pigmentation, Response To Oxidative Stress, Stem Cell Differentiation
Quite a number of genes have been found to play important roles in Generalized Vitiligo, such as CAT, CD4, CD8A, CTLA4, DCT, FOXD3, HLA-A, HLA-DQA1, HLA-E, IFNG, IL2, IL4, NLRP1, NOD2, POMC, PTPN22, SH2D1A, TNF, TYR, XBP1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.