This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Friedreich Ataxia
Information about Friedreich Ataxia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Friedreich Ataxia
Most recent studies have shown that Friedreich Ataxia shares some biological mechanisms with abnormal-degeneration, abnormal-degeneration, ataxia, ataxia, ataxia-spinocerebellar, ataxia-spinocerebellar, atrophy, atrophy, cardiomyopathies, cardiomyopathies, cerebellar-ataxia, cerebellar-ataxia, diabetes-mellitus, diabetes-mellitus, friedreich-ataxia, heart-diseases, hereditary-ataxia-unspecified, hereditary-ataxia-unspecified, huntington-disease, huntington-disease, hypertrophic-cardiomyopathy, hypertrophic-cardiomyopathy, hypertrophy, hypertrophy, muscular-atrophy, nerve-degeneration, nervous-system-disorder, nervous-system-disorder, nervousness, nervousness, neurodegenerative-disorders, neurodegenerative-disorders, spinocerebellar-degeneration, spinocerebellar-degeneration, trinucleotide-repeat-expansion, trinucleotide-repeat-expansion, vitamin-e-deficiency.
Among the many pathways, these few ones have gauged particular interests from scientists studying Friedreich Ataxia, and have been seen in publications frequently: Aging, Aging, Cell Cycle, Cell Death, Cell Death, Cognition, Cognition, Dna Methylation, Dna Repair, Dna Repair, Dna Replication, Electron Transport, Electron Transport, Electron Transport Chain, Electron Transport Chain, Excretion, Gene Silencing, Gene Silencing, Hypersensitivity, Hypersensitivity, Iron-sulfur Cluster Assembly, Iron-sulfur Cluster Assembly, Localization, Localization, Methylation, Methylation, Mismatch Repair, Oxidative Phosphorylation, Oxidative Phosphorylation, Pathogenesis, Pathogenesis, Pyruvate Oxidation, Reflex, Reflex, Regeneration, Rna Interference, Secretion, Secretion, Transport, Transport
Quite a number of genes have been found to play important roles in Friedreich Ataxia, such as ACO1, ACO1, APBA1, APBA1, APTX, APTX, ATN1, ATXN1, ATXN1, ATXN2, ATXN2, ATXN3, ATXN3, ATXN7, CACNA1A, DLD, DLD, EPO, EPO, FANCA, FANCA, FARSA, FTMT, FTMT, FXN, FXN, GAA, GAA, INS, LY6E, LY6E, PES1, SETX, TTPA, TTPA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.