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Disease Info Card
Encephalomyelitis
Information about Encephalomyelitis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Encephalomyelitis
Most recent studies have shown that Encephalomyelitis shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, autoimmunity, cns-disorder, demyelinating-diseases, demyelination, encephalitis, encephalomyelitis-acute-disseminated, encephalomyelitis-equine, experimental-autoimmune-encephalomyelitis, infective-disorder, inflammation, multiple-sclerosis, neoplasms, nervous-system-disorder, nervousness, sclerosis, venezuelan-equine-encephalomyelitis, virus-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Encephalomyelitis, and have been seen in publications frequently: Cell Activation, Cell Adhesion, Cell Death, Cell Differentiation, Cell Proliferation, Cytokine Production, Cytokine Secretion, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Neuroprotection, Pathogenesis, Secretion, Sensitization, T Cell Activation, T Cell Proliferation, Tolerance Induction, Translation, Viral Replication
Quite a number of genes have been found to play important roles in Encephalomyelitis, such as CD4, CSF2, CTLA4, IFNG, IL10, IL17A, IL2, IL4, IL6, LAMC2, MAG, MBL2, MBP, MOG, PDXP, PLP1, PRDX5, PRG2, PTHLH, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.