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Disease Info Card
Deaf Mutism
Information about Deaf Mutism: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Deaf Mutism
Most recent studies have shown that Deaf Mutism shares some biological mechanisms with complete-hearing-loss, congenital-abnormality, congenital-hypothyroidism, deafness-congenital, diabetes-mellitus-insulin-dependent, endemic-cretinism, endemic-goiter, goiter, hearing-problem, hypothyroidism, mental-deficiency, mutism, pendreds-syndrome, pregnancy-complications-infectious, retinitis-pigmentosa, rubella, sensorineural-hearing-loss-(disorder).
Among the many pathways, these few ones have gauged particular interests from scientists studying Deaf Mutism, and have been seen in publications frequently: Blood Circulation, Bone Maturation, Brain Development, Cell Death, Cellular Homeostasis, Chloride Transport, Echolocation, Embryo Development, Excretion, Localization, Mating, Membrane Fusion, Ossification, Pathogenesis, Reflex, Secretion, Short-term Memory, Translation, Transport
Quite a number of genes have been found to play important roles in Deaf Mutism, such as ABR, COL9A3, DGCR2, GJB2, GJB6, GJD2, MYO15A, MYO7A, NDUFB6, OTOF, PCDH15, PRB4, SCN5A, SLC26A4, TMIE, TMPRSS3, TRH. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.