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Disease Info Card
Corneal Neovascularization
Information about Corneal Neovascularization: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Corneal Neovascularization
Most recent studies have shown that Corneal Neovascularization shares some biological mechanisms with blind-vision, chemical-burns, cicatrix, conjunctival-diseases, corneal-diseases, corneal-edema, corneal-ulcer, disorder-of-eye, edema, eye-burns, inflammation, keratitis, keratitis-herpetic, malignant-neoplasms, neoplasms, pathologic-neovascularization, tumor-angiogenesis, ulcer.
Among the many pathways, these few ones have gauged particular interests from scientists studying Corneal Neovascularization, and have been seen in publications frequently: Angiogenesis, Cell Growth, Cell Migration, Cell Proliferation, Chemotaxis, Endothelial Cell Migration, Endothelial Cell Proliferation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Lymphangiogenesis, Pathogenesis, Pigmentation, Regeneration, Reverse Transcription, Secretion, Tube Formation, Vasculogenesis, Wound Healing
Quite a number of genes have been found to play important roles in Corneal Neovascularization, such as ANGPT2, COL18A1, FGF2, FLT1, HBA1, IL6, KDR, LYVE1, MMP2, MMP9, PECAM1, PLG, SERPINF1, THBS1, TNF, VEGFA, VEGFC. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.