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Disease Info Card
Eye Burns
Information about Eye Burns: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Eye Burns
Most recent studies have shown that Eye Burns shares some biological mechanisms with blind-vision, cataract, chemical-burns, cicatrix, conjunctival-diseases, corneal-burn, corneal-diseases, corneal-neovascularization, corneal-ulcer, disorder-of-eye, eye-foreign-bodies, eye-injuries, glaucoma, inflammation, keratitis, pathologic-neovascularization, scleral-diseases, thermal-burn, ulcer.
Among the many pathways, these few ones have gauged particular interests from scientists studying Eye Burns, and have been seen in publications frequently: Angiogenesis, Blood Circulation, Cell Adhesion, Cell Migration, Cell Proliferation, Coagulation, Enucleation, Inflammatory Response, Localization, Lymphangiogenesis, Pathogenesis, Pigmentation, Proteolysis, Regeneration, Secretion, Sensitization, Tear Secretion, Transdifferentiation, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Eye Burns, such as AMT, ARC, CKAP4, EGF, FN1, IL6, KRT3, MMP2, MMP9, NOL3, PLA2G2D, PLG, RPE65, SERPINF1, TNF, TNFSF14, TP63, UVRAG, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.