Disease Info Card

Cold Ischemia

Information about Cold Ischemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Cold Ischemia

Most recent studies have shown that Cold Ischemia shares some biological mechanisms with decreased-immunologic-activity-[pe], delayed-graft-function, diabetes-mellitus, edema, flushing, graft-failure, hemorrhage, inflammation, ischemia, kidney-diseases, kidney-failure, kidney-failure-chronic, liver-diseases, reperfusion-injury, stenosis, thrombosis, warm-ischemia.

Among the many pathways, these few ones have gauged particular interests from scientists studying Cold Ischemia, and have been seen in publications frequently: Aging, Cell Activation, Cell Death, Coagulation, Diuresis, Donor Selection, Excretion, Glomerular Filtration, Glycolysis, Immune Response, Inflammatory Response, Insulin Secretion, Liver Regeneration, Localization, Pathogenesis, Regeneration, Secretion, Sensitization, Transport, Vasoconstriction

Quite a number of genes have been found to play important roles in Cold Ischemia, such as CASP3, CIT, DHDDS, ECD, EPHB4, F2, GNL3, HLA-A, HLA-DRB4, HMOX1, IL2, IL6, INS, KRAS, MPO, NOS2, S100A6, SLC17A5, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Cold Ischemia Related Genes

click to see detail information for each gene

CASP3 CIT DHDDS
ECD EPHB4 F2
GNL3 HLA-A HLA-DRB4
HMOX1 IL2 IL6
INS KRAS MPO
NOS2 S100A6 SLC17A5
TNF