Disease Info Card

Chronic Stable Angina

Information about Chronic Stable Angina: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Chronic Stable Angina

Most recent studies have shown that Chronic Stable Angina shares some biological mechanisms with acute-coronary-syndrome, acute-myocardial-infarction, angina-pectoris, angina-unstable, atherosclerosis, coronary-artery-disease, coronary-heart-disease, coronary-stenosis, dental-plaque, depressive-disorder, diabetes-mellitus, heart-diseases, heart-failure, hypertensive-disease, infarction, ischemia, myocardial-infarction, myocardial-ischemia, pain, stenosis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Chronic Stable Angina, and have been seen in publications frequently: Angiogenesis, Cell Activation, Cell Adhesion, Coagulation, Excretion, Fatty Acid Oxidation, Fibrinolysis, Glomerular Filtration, Hemostasis, Immune Response, Inflammatory Response, Localization, Neutrophil Activation, Pathogenesis, Platelet Activation, Platelet Aggregation, Reflex, Secretion, Vasoconstriction, Vasodilation

Quite a number of genes have been found to play important roles in Chronic Stable Angina, such as ACCS, ACE, ACSS2, APCS, BIRC6, CRP, CSRP1, F3, GNL3, IL10, IL6, KRAS, MITF, MMP9, PLA2G15, SELP, SERPINA5, SH2D1A, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.