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Disease Info Card
Angina, Unstable
Information about Angina, Unstable: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Angina, Unstable
Most recent studies have shown that Angina, Unstable shares some biological mechanisms with acute-coronary-syndrome, acute-myocardial-infarction, angina-pectoris, cerebrovascular-accident, chest-pain, chronic-stable-angina, coronary-artery-disease, coronary-heart-disease, dental-plaque, diabetes-mellitus, heart-diseases, heart-failure, hemorrhage, infarction, ischemia, myocardial-infarction, myocardial-ischemia, pain, stenosis, thrombosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Angina, Unstable, and have been seen in publications frequently: Aging, Angiogenesis, Blood Coagulation, Cell Activation, Cell Adhesion, Coagulation, Excretion, Fibrinolysis, Glomerular Filtration, Hemostasis, Immune Response, Inflammatory Response, Localization, Pathogenesis, Platelet Activation, Platelet Aggregation, Secretion, Transport, Vasoconstriction, Vasodilation
Quite a number of genes have been found to play important roles in Angina, Unstable, such as ACACA, ACCS, ACE, ACSS2, CHKA, CHKB, CRP, CSRP1, GNL3, IL6, KRAS, MB, MITF, PLA2G15, SELP, SERPINA5, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.