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- Table of Contents
Information about Childhood Acute Myeloid Leukemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Childhood Acute Myeloid Leukemia shares some biological mechanisms with acute-leukemia, acute-lymphocytic-leukemia, acute-monocytic-leukemia, acute-myelomonocytic-leukemia, acute-promyelocytic-leukemia, adult-acute-myeloblastic-leukemia, chromosomal-translocation, cytogenetic-abnormality, down-syndrome, dysmyelopoietic-syndromes, hematologic-neoplasms, l1-acute-lymphoblastic-leukemia, leukemia, leukemia-myelocytic-acute, malignant-neoplasms, malignant-paraganglionic-neoplasm, myeloblastic-leukemia, myeloid-leukemia, neoplasm-residual, neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Childhood Acute Myeloid Leukemia, and have been seen in publications frequently: Cell Adhesion, Cell Cycle, Cell Cycle Checkpoint, Cell Death, Cell Differentiation, Cell Growth, Cell Migration, Cell Proliferation, Dna Methylation, Drug Resistance, Induction Of Apoptosis, Interphase, Localization, Methylation, Neutrophil Differentiation, Pathogenesis, Programmed Cell Death, Reverse Transcription, Translation, Transport
Quite a number of genes have been found to play important roles in Childhood Acute Myeloid Leukemia, such as CD33, CD34, CR1, CTLA4, EFS, FANCB, FLT3, HLA-DQA1, INVS, KIT, LMLN, MID1, NOD2, NPM1, RUNX1, RUNX1T1, WT1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.