Disease Info Card

Myeloid Leukemia

Information about Myeloid Leukemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Myeloid Leukemia

Most recent studies have shown that Myeloid Leukemia shares some biological mechanisms with acute-leukemia, acute-lymphocytic-leukemia, acute-promyelocytic-leukemia, anemia, blast-phase, cytogenetic-abnormality, dysmyelopoietic-syndromes, graft-vs-host-disease, hematologic-neoplasms, leukemia, leukemia-myelocytic-acute, lymphoid-leukemia, lymphoma, malignant-neoplasms, malignant-paraganglionic-neoplasm, myeloid-leukemia-chronic, myeloproliferative-disease, neoplasms, philadelphia-chromosome.

Among the many pathways, these few ones have gauged particular interests from scientists studying Myeloid Leukemia, and have been seen in publications frequently: Angiogenesis, Cell Cycle, Cell Cycle Arrest, Cell Death, Cell Differentiation, Cell Growth, Cell Proliferation, Coagulation, Dna Methylation, Drug Resistance, Immune Response, Induction Of Apoptosis, Interphase, Localization, Metaphase, Methylation, Pathogenesis, Reverse Transcription, Secretion, Transport

Quite a number of genes have been found to play important roles in Myeloid Leukemia, such as ABL1, ASXL1, BCR, CD33, CD34, CSF2, CSF3, CTLA4, FANCB, FLT3, HLA-DQA1, IL3, KIT, MTTP, NOD2, PAFAH1B1, RUNX1, SF3B1, TNF, YWHAE. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Myeloid Leukemia Related Genes

click to see detail information for each gene

ABL1 ASXL1 BCR
CD33 CD34 CSF2
CSF3 CTLA4 FANCB
FLT3 HLA-DQA1 IL3
KIT MTTP NOD2
PAFAH1B1 RUNX1 SF3B1
TNF YWHAE