Disease Info Card

Brugada Syndrome (disorder)

Information about Brugada Syndrome (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Brugada Syndrome (disorder)

Most recent studies have shown that Brugada Syndrome (disorder) shares some biological mechanisms with arrhythmogenic-right-ventricular-dysplasia, atrial-fibrillation, bundle-branch-block, cardiac-arrest, cardiac-arrhythmia, cardiac-death, cardiac-fibrillation, cardiomyopathies, channelopathies, heart-diseases, hypertrophy, long-qt-syndrome, malignant-paraganglionic-neoplasm, polymorphic-ventricular-tachycardia, right-bundle-branch-block, sudden-cardiac-death, sudden-death, syncope, tachycardia-ventricular, ventricular-fibrillation.

Among the many pathways, these few ones have gauged particular interests from scientists studying Brugada Syndrome (disorder), and have been seen in publications frequently: Aging, Cardiac Conduction, Cell Adhesion, Cellular Localization, Heart Development, Hypersensitivity, Innervation, Insulin Secretion, Localization, Membrane Depolarization, Membrane Organization, Muscle Contraction, Pathogenesis, Reflex, Rna Processing, Secretion, Translation, Transpiration, Transport, Transposition

Quite a number of genes have been found to play important roles in Brugada Syndrome (disorder), such as BLM, GP1BB, GPD1L, IGKV1-5, KCND3, KCNE1, KCNH2, KCNQ1, NAMPT, PGR, SCD, SCN1B, SCN5A, TMEM37, UBN1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.