Disease Info Card

Arthrogryposis Multiplex Congenita

Information about Arthrogryposis Multiplex Congenita: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Arthrogryposis Multiplex Congenita

Most recent studies have shown that Arthrogryposis Multiplex Congenita shares some biological mechanisms with arthrogryposis, atrophy, congenital-abnormality, congenital-clubfoot, dislocations, dystrophy, flexed-fetal-attitude, hip-dislocation-congenital, hypoplasia, muscle-contracture, muscular-atrophy, muscular-dystrophy, myasthenia-gravis, myasthenias, myopathy, nervousness, neuromuscular-diseases, pregnancy-complications, scoliosis-unspecified, weakness.

Among the many pathways, these few ones have gauged particular interests from scientists studying Arthrogryposis Multiplex Congenita, and have been seen in publications frequently: Brain Development, Cell Development, Cell Differentiation, Immune Response, Innervation, Insemination, Lipid Storage, Localization, Membrane Fusion, Muscle Atrophy, Myelination, Oxidative Phosphorylation, Pathogenesis, Reflex, Regeneration, Schwann Cell Development, Schwann Cell Differentiation, Secretion, Transposition

Quite a number of genes have been found to play important roles in Arthrogryposis Multiplex Congenita, such as ARC, CHRNA4, FLAD1, HRAS, KCNH2, KCNJ11, MAG, MPZ, NAIP, NOL3, PES1, PLOD2, PSMA7, RAPSN, SMN1, SNRPN, SYNE1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.