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Disease Info Card
Arachnodactyly
Information about Arachnodactyly: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Arachnodactyly
Most recent studies have shown that Arachnodactyly shares some biological mechanisms with acquired-kyphosis, aneurysm, aortic-aneurysm, blepharophimosis, blepharoptosis, congenital-abnormality, congenital-camptodactyly, congenital-contractural-arachnodactyly, congenital-ectopia, connective-tissue-diseases, ectopia-lentis, hypoplasia, kyphoscoliosis-deformity-of-spine, marfan-syndrome, marfanoid-joint-hypermobility-syndrome, mitral-valve-prolapse-syndrome, muscle-contracture, pathological-dilatation, scoliosis-unspecified.
Among the many pathways, these few ones have gauged particular interests from scientists studying Arachnodactyly, and have been seen in publications frequently: Bone Maturation, Bone Resorption, Chondrocyte Differentiation, Fibroblast Proliferation, Keratinization, Localization, Meiosis Ii, Muscle Atrophy, Myelination, Neural Tube Closure, Ossification, Osteoblast Differentiation, Osteoblast Proliferation, Pathogenesis, Pigmentation, Regulation Of Organ Formation, Reverse Transcription, Secretion, Tendon Development, Translation
Quite a number of genes have been found to play important roles in Arachnodactyly, such as CTSC, EFEMP2, EGF, ELN, FBN1, FBN2, FBN3, INVS, LMLN, MFAP5, MTHFR, MTM1, PES1, SCARF2, SNRPN, TGFBR1, TGFBR2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.