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Disease Info Card
Congenital Camptodactyly
Information about Congenital Camptodactyly: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Congenital Camptodactyly
Most recent studies have shown that Congenital Camptodactyly shares some biological mechanisms with arthrogryposis, arthropathy, blepharoptosis, cleft-palate, clinodactyly, congenital-abnormality, congenital-clubfoot, congenital-hand-deformities, developmental-delay-(disorder), dwarfism, dysplasia, flexed-fetal-attitude, growth-retardation, hypoplasia, microcephaly, micrognathism, muscle-contracture, scoliosis-unspecified, syndactyly.
Among the many pathways, these few ones have gauged particular interests from scientists studying Congenital Camptodactyly, and have been seen in publications frequently: Brain Development, Cell Death, Cognition, Endochondral Bone Growth, Glycosylation, Granuloma Formation, Limb Development, Limb Morphogenesis, Mating, Methylation, Muscle Contraction, Myelination, Ossification, Pathogenesis, Pigmentation, Programmed Cell Death, Secretion, Sex Determination, Translation, Transposition
Quite a number of genes have been found to play important roles in Congenital Camptodactyly, such as CRLF1, EVX2, FBN2, HOXD13, INVS, LIFR, LMLN, MB, MYH3, NOD2, NSD1, PNPLA6, PRG4, S100A10, TGFBR1, TGFBR2, TNNI2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.