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Disease Info Card
Primary Systemic Amyloidosis
Information about Primary Systemic Amyloidosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Primary Systemic Amyloidosis
Most recent studies have shown that Primary Systemic Amyloidosis shares some biological mechanisms with amyloid-deposition, amyloid-neuropathies, amyloidosis, amyloidosis-familial, cardiomyopathies, heart-failure, hemorrhage, kidney-diseases, kidney-failure, multiple-myeloma, neoplasms, nephrotic-syndrome, plaque-amyloid, polyneuropathy, primary-amyloidosis, proteinuria-of-undiagnosed-cause, secondary-amyloidosis, senile-cardiac-amyloidosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Primary Systemic Amyloidosis, and have been seen in publications frequently: Acute-phase Response, Aging, Amyloid Fibril Formation, Cell Death, Cell Proliferation, Coagulation, Excretion, Glomerular Filtration, Hemostasis, Inflammatory Response, Interphase, Localization, Pathogenesis, Pigmentation, Protein Folding, Proteolysis, Reflex, Secretion, Transport, Tropism
Quite a number of genes have been found to play important roles in Primary Systemic Amyloidosis, such as ALB, APCS, APOA1, APOA2, CALR, CEL, CRP, FAP, GLMN, GSN, LYZ, MEFV, MYOM2, SH2D1A, TEAD1, TRIM21, TTR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.