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Disease Info Card
Acantholysis
Information about Acantholysis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Acantholysis
Most recent studies have shown that Acantholysis shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, bulla, bullous-pemphigoid, carcinoma, dermatitis, dermatologic-disorders, dyskeratosis, exanthema, familial-benign-pemphigus, keratosis, keratosis-follicularis, malignant-squamous-cell-neoplasm, neoplasms, pemphigus-foliaceus, skin-neoplasms, tissue-adhesions, transient-acantholytic-dermatosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Acantholysis, and have been seen in publications frequently: Anagen, Cell Adhesion, Cell Cycle, Cell Death, Cell-cell Adhesion, Complement Activation, Cytolysis, Endocytosis, Exocytosis, Immune Response, Intracellular Signal Transduction, Keratinization, Localization, Pathogenesis, Pigmentation, Programmed Cell Death, Proteolysis, Secretion, Telogen, Wound Healing
Quite a number of genes have been found to play important roles in Acantholysis, such as AKR1C2, ATP2A2, ATP2C1, C3, CDH1, DSG1, DSG3, DSP, DSPP, DST, IL10, IL4, KRT14, KRT5, PLAU, PLG, PPL, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.