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Disease Info Card
Familial Benign Pemphigus
Information about Familial Benign Pemphigus: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Familial Benign Pemphigus
Most recent studies have shown that Familial Benign Pemphigus shares some biological mechanisms with acantholysis, autoimmune-reaction, bulla, bullous-pemphigoid, carcinoma, dermatitis, dermatologic-disorders, dyskeratosis, exanthema, keratosis, keratosis-follicularis, malignant-squamous-cell-neoplasm, neoplasms, psoriasis, skin-diseases-genetic, tissue-adhesions, transient-acantholytic-dermatosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Familial Benign Pemphigus, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Growth, Desmosome Assembly, Hypersensitivity, Immune Response, Ion Transport, Keratinization, Keratinocyte Differentiation, Localization, Pathogenesis, Proteolysis, Relaxation Of Vascular Smooth Muscle, Response To Vitamin, Secretion, Secretory Pathway, Steroid Hormone Secretion, Translation, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Familial Benign Pemphigus, such as AKR1C2, AMY2A, ATP2A2, ATP2B2, ATP2C1, ATP2C2, BLOC1S6, C2, CDH1, DSG1, DSG3, DSP, F7, IVL, MTSS1, PLAU, PLG, PRH1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.