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Disease Info Card
Keratosis Follicularis
Information about Keratosis Follicularis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Keratosis Follicularis
Most recent studies have shown that Keratosis Follicularis shares some biological mechanisms with acantholysis, carcinoma, common-wart, dermatitis, dermatologic-disorders, dyskeratosis, exanthema, familial-benign-pemphigus, hyperkeratosis, hyperkeratosis-follicularis-et-parafollicularis, ichthyoses, keratosis, melanocytic-nevus, neoplasms, palmoplantar-keratosis, psoriasis, skin-diseases-genetic, skin-neoplasms, tissue-adhesions.
Among the many pathways, these few ones have gauged particular interests from scientists studying Keratosis Follicularis, and have been seen in publications frequently: Anagen, Cell Adhesion, Cell Cycle, Cell Proliferation, Cell-cell Adhesion, Cornification, Excretion, Exocytosis, Glycosylation, Hypersensitivity, Inflammatory Response, Keratinization, Keratinocyte Differentiation, Localization, Pathogenesis, Pigmentation, Secretion, Secretory Pathway, Skin Development, Transport
Quite a number of genes have been found to play important roles in Keratosis Follicularis, such as AKR1C2, ATP2A1, ATP2A2, ATP2A3, ATP2C1, CALR, CDH1, DSG1, DSP, DSPP, IVL, KRT1, KRT10, KRT16, KRT17, PLA2G1B, PLG, PTGDR, SAT1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.