Disease Info Card

Abnormal Involuntary Movement

Information about Abnormal Involuntary Movement: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Abnormal Involuntary Movement

Most recent studies have shown that Abnormal Involuntary Movement shares some biological mechanisms with akathisia, basal-ganglia-diseases, chorea, chronic-schizophrenia, depressive-disorder, dyskinesia-drug-induced, dyskinetic-syndrome, dystonia-disorders, extrapyramidal-sign, huntington-disease, involuntary-movements, lingual-facial-buccal-dyskinesia, mental-disorders, mood-disorders, movement-disorders, parkinson-disease, psychotic-disorders, schizoaffective-disorder, schizophrenia, secondary-parkinson-disease.

Among the many pathways, these few ones have gauged particular interests from scientists studying Abnormal Involuntary Movement, and have been seen in publications frequently: Aging, Cognition, Electron Transport, Electron Transport Chain, Habituation, Hyperphosphorylation, Hypersensitivity, Innervation, Localization, Locomotion, Neuroprotection, Pathogenesis, Reflex, Response To Amphetamine, Response To Clozapine, Sensitization, Synaptic Transmission, Translation, Transport, Turning Behavior

Quite a number of genes have been found to play important roles in Abnormal Involuntary Movement, such as BBS9, COMT, CYP2D6, DIO2, DRD2, DRD3, EEF1A2, FOS, FOSB, GRM5, HTR1A, LMOD1, LRP2, MCF2L, NANS, PDYN, PRL, TH, TSPAN31. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.