Disease Info Card

Extrapyramidal Sign

Information about Extrapyramidal Sign: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Extrapyramidal Sign

Most recent studies have shown that Extrapyramidal Sign shares some biological mechanisms with agitation, akathisia, alzheimers-disease, basal-ganglia-diseases, bipolar-disorder, chronic-schizophrenia, dementia, depressive-disorder, dyskinesia-drug-induced, dyskinetic-syndrome, dystonia-disorders, lingual-facial-buccal-dyskinesia, manic, mental-disorders, movement-disorders, parkinson-disease, psychotic-disorders, schizoaffective-disorder, schizophrenia, secondary-parkinson-disease.

Among the many pathways, these few ones have gauged particular interests from scientists studying Extrapyramidal Sign, and have been seen in publications frequently: Aging, Brain Development, Cardiac Conduction, Cognition, Excretion, Hypersensitivity, Innervation, Lactation, Oxidative Phosphorylation, Pathogenesis, Prepulse Inhibition, Reflex, Response To Clozapine, Response To Risperidone, Secretion, Startle Response, Swimming, Translation, Transport

Quite a number of genes have been found to play important roles in Extrapyramidal Sign, such as ARNTL, BBS9, BCHE, CSF2, CYP2D6, DIO2, DIO3, DRD2, FGF9, GDI1, HTR1A, HTR2A, LAMC2, LMOD1, NANS, PRL, PRNP, TSPAN31, USH1G. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.