pathway Info Card

Neutrophil Extravasation

Information about Neutrophil Extravasation: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Neutrophil Extravasation

Most recent studies have shown that Neutrophil Extravasation shares some biological mechanisms with acute-inflammatory-response, cell-activation, cell-adhesion, cell-migration, chemokine-production, chemotaxis, complement-activation, diapedesis, endothelial-cell-activation, immune-response, inflammatory-response, leukocyte-chemotaxis, localization, mast-cell-activation, neutrophil-activation, pathogenesis, phagocytosis, secretion, transport, wound-healing.

Among the many pathways, these few ones have gauged particular interests from scientists studying Neutrophil Extravasation, and have been seen in publications frequently: acute-inflammatory-response, cell-activation, cell-adhesion, cell-migration, chemokine-production, chemotaxis, complement-activation, diapedesis, endothelial-cell-activation, immune-response, inflammatory-response, leukocyte-chemotaxis, localization, mast-cell-activation, neutrophil-activation, pathogenesis, phagocytosis, secretion, transport, wound-healing

Quite a number of genes have been found to play important roles in Neutrophil Extravasation, such as ANXA1, CALCA, CCL2, CTLA4, ELANE, FPR1, HLA-DQA1, ICAM1, IL6, ITGAM, ITGB2, LTBR, MPO, NOD2, SELE, SELL, SELP, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this pathway. Plesae stay updated.