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- Table of Contents
Information about X-linked Severe Combined Immunodeficiency: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that X-linked Severe Combined Immunodeficiency shares some biological mechanisms with agammaglobulinemia, congenital-combined-immunodeficiency, dog-diseases, genetic-diseases-x-linked, graft-vs-host-disease, granulomatous-disease-chronic, granulomatous-disorder, hereditary-diseases, immunologic-deficiency-syndromes, infective-disorder, leukemia, leukemogenesis, malignant-neoplasms, neoplasms, primary-immune-deficiency-disorder, severe-combined-immunodeficiency, wiskott-aldrich-syndrome, x-linked-agammaglobulinemia, x-linked-combined-immunodeficiency-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying X-linked Severe Combined Immunodeficiency, and have been seen in publications frequently: Cell Development, Cell Differentiation, Cell Growth, Cell Proliferation, Endocytosis, Immune Response, Immunoglobulin Production, Intracellular Signal Transduction, Isotype Switching, Localization, Lymphocyte Differentiation, Lymphocyte Proliferation, Methylation, Natural Killer Cell Differentiation, Oncogenesis, Pathogenesis, Regeneration, Response To Interleukin-2, T Cell Proliferation, Translation
Quite a number of genes have been found to play important roles in X-linked Severe Combined Immunodeficiency, such as ADA, CD34, CD40LG, FUT1, IL15, IL17C, IL2, IL21, IL21R, IL22, IL2RG, IL4, IL7, IL9, JAK1, JAK3, LMO2, WAS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.