This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Granulomatous Disease, Chronic
Information about Granulomatous Disease, Chronic: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Granulomatous Disease, Chronic
Most recent studies have shown that Granulomatous Disease, Chronic shares some biological mechanisms with abscess, aspergillosis, bacterial-infections, granuloma, granulomatous-disorder, immunologic-deficiency-syndromes, infective-disorder, inflammation, liver-abscess, lung-diseases-fungal, lymphadenitis, mycoses, neoplasms, phagocyte-bactericidal-dysfunction, pneumonia, primary-immune-deficiency-disorder, sarcoidosis, severe-combined-immunodeficiency, tuberculosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Granulomatous Disease, Chronic, and have been seen in publications frequently: Antibody-dependent Cellular Cytotoxicity, Cell Activation, Cell Death, Chemotaxis, Cytolysis, Electron Transport, Granuloma Formation, Hypersensitivity, Immune Response, Inflammatory Response, Localization, Neutrophil Activation, Neutrophil Chemotaxis, Pathogenesis, Phagocytosis, Protein Phosphorylation, Respiratory Burst, Secretion, Transport, Virulence
Quite a number of genes have been found to play important roles in Granulomatous Disease, Chronic, such as AKAP13, CAT, CD33, CLEC11A, CYBA, CYBB, IFNG, ING1, METAP2, MPO, NCF1, NCF2, NSFL1C, PLEK, STXBP1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.