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Disease Info Card
Vitelliform Macular Dystrophy
Information about Vitelliform Macular Dystrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Vitelliform Macular Dystrophy
Most recent studies have shown that Vitelliform Macular Dystrophy shares some biological mechanisms with abnormal-degeneration, adult-onset-vitelliform-macular-dystrophy, age-related-macular-degeneration, atrophy, blind-vision, choroidal-neovascularization, dystrophy, hereditary-corneal-dystrophy, macule, maculopathy, pathologic-neovascularization, retinal-degeneration, retinal-detachment, retinal-diseases, retinal-dystrophies, retinitis-pigmentosa, stargardts-disease, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Vitelliform Macular Dystrophy, and have been seen in publications frequently: Aging, Cell Proliferation, Cellular Localization, Contact Inhibition, Fibrinolysis, Fluid Transport, Localization, Mrna Splicing, Oxygen Homeostasis, Pathogenesis, Phagocytosis, Phototransduction, Pigment Accumulation, Platelet Activation, Platelet Aggregation, Reflex, Regeneration, Segmentation, Translation, Transport
Quite a number of genes have been found to play important roles in Vitelliform Macular Dystrophy, such as ABCA4, BEST1, COX5A, DMD, ERG, KCNH2, MID1, MITF, PLXNA2, PRPH, PRPH2, RPE, SCGB1A1, SEPSECS, TIMP3, TNFSF14, USP9X, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.