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Disease Info Card
Choroidal Neovascularization
Information about Choroidal Neovascularization: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Choroidal Neovascularization
Most recent studies have shown that Choroidal Neovascularization shares some biological mechanisms with age-related-macular-degeneration, atrophy, blind-vision, choroidal-neovascular-membrane-(disorder), diabetic-retinopathy, edema, exudative-age-related-macular-degeneration, hemorrhage, inflammation, macular-retinal-edema, macule, myopia, myopia-degenerative, pathologic-neovascularization, retinal-detachment, retinal-diseases, retinal-neovascularization, tumor-angiogenesis, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Choroidal Neovascularization, and have been seen in publications frequently: Aging, Angiogenesis, Cell Adhesion, Cell Death, Cell Migration, Cell Proliferation, Coagulation, Complement Activation, Endothelial Cell Migration, Endothelial Cell Proliferation, Enucleation, Inflammatory Response, Localization, Pathogenesis, Pigmentation, Secretion, Segmentation, Transport, Tube Formation, Wound Healing
Quite a number of genes have been found to play important roles in Choroidal Neovascularization, such as AMD1, CCL2, CFH, COX5A, FANCA, FGF2, FLNB, FXN, NXF1, OCA2, PEA15, PLXNA2, PNPLA2, RPE, SERPINF1, SLC2A1, TNF, USO1, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.