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Disease Info Card
Uveal Neoplasms
Information about Uveal Neoplasms: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Uveal Neoplasms
Most recent studies have shown that Uveal Neoplasms shares some biological mechanisms with cell-invasion, choroid-neoplasms, ciliary-body-melanoma, cutaneous-melanoma, dental-plaque, eye-neoplasms, glaucoma, iris-diseases, liver-neoplasms, malignant-neoplasms, malignant-paraganglionic-neoplasm, melanocytic-nevus, melanoma, neoplasm-metastasis, neoplasms, ocular-melanoma, scleral-diseases, skin-neoplasms, uveal-diseases, uveal-melanoma.
Among the many pathways, these few ones have gauged particular interests from scientists studying Uveal Neoplasms, and have been seen in publications frequently: Angiogenesis, Cell Adhesion, Cell Cycle, Cell Cycle Arrest, Cell Death, Cell Division, Cell Growth, Cell Migration, Cell Proliferation, Coagulation, Cytolysis, Enucleation, Hypersensitivity, Immune Response, Localization, Methylation, Pathogenesis, Pigmentation, Reverse Transcription, Secretion
Quite a number of genes have been found to play important roles in Uveal Neoplasms, such as BRAF, BRCA1, CDKN2A, CTLA4, GNAQ, HGF, HLA-DQA1, IL2, IL6, KIT, MAPK1, MAPK3, MLANA, NOD2, SLC25A5, SP6, TP53, TYR, VEGFA, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.