This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Unverricht-lundborg Syndrome
Information about Unverricht-lundborg Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Unverricht-lundborg Syndrome
Most recent studies have shown that Unverricht-lundborg Syndrome shares some biological mechanisms with action-myoclonus, ataxia, atrophy, dementia, epilepsies-myoclonic, epilepsy, epilepsy-generalized, herpes-zoster-oticus, lafora-disease, merrf-syndrome, myoclonic-epilepsies-progressive, myoclonic-epilepsy-juvenile, myoclonus, nervousness, neurodegenerative-disorders, neuronal-ceroid-lipofuscinoses, tonic-clonic-epilepsy, type-i-mucolipidosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Unverricht-lundborg Syndrome, and have been seen in publications frequently: Aging, Cell Cycle, Cell Death, Cognition, Dna Deamination, Electron Transport, Electron Transport Chain, Excretion, Heteroduplex Formation, Intestinal Absorption, Localization, Methylation, Mrna Transcription, Muscle Contraction, Neuroprotection, Pathogenesis, Proteolysis, Reflex, S Phase, Transport
Quite a number of genes have been found to play important roles in Unverricht-lundborg Syndrome, such as CHRNA4, CLN3, CSTB, CTSB, CTSK, CTSS, EPM2A, KCNQ2, KCNQ3, MCAT, NHLRC1, PFKL, PRICKLE1, PYCARD, SCN1A, TYMS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.