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Disease Info Card
Neuronal Ceroid-lipofuscinoses
Information about Neuronal Ceroid-lipofuscinoses: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Neuronal Ceroid-lipofuscinoses
Most recent studies have shown that Neuronal Ceroid-lipofuscinoses shares some biological mechanisms with adult-neuronal-ceroid-lipofuscinosis, atrophy, blind-vision, dementia, dog-diseases, epilepsies-myoclonic, epilepsy, infantile-neuronal-ceroid-lipofuscinosis, juvenile-neuronal-ceroid-lipfuscinosis, late-infantile-neuronal-ceroid-lipfuscinosis, lipoidosis, lysosomal-storage-diseases, myoclonus, nerve-degeneration, nervousness, neurodegenerative-disorders, storage-disease, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Neuronal Ceroid-lipofuscinoses, and have been seen in publications frequently: Aging, Autophagy, Brain Development, Cell Death, Cell Growth, Cell Proliferation, Endocytosis, Excretion, Glycosylation, Immune Response, Localization, Methylation, Neuron Death, Pathogenesis, Phagocytosis, Pigmentation, Proteolysis, Reflex, Translation, Transport
Quite a number of genes have been found to play important roles in Neuronal Ceroid-lipofuscinoses, such as ACD, CLN3, CLN5, CLN6, CLN8, CTSD, DNAJC5, ERG, KCNH2, MFSD8, NCL, PPP5C, PPT1, TAC1, TPP1, TTPA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.