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Disease Info Card
Myoclonic Epilepsies, Progressive
Information about Myoclonic Epilepsies, Progressive: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Myoclonic Epilepsies, Progressive
Most recent studies have shown that Myoclonic Epilepsies, Progressive shares some biological mechanisms with ataxia, ataxia-spinocerebellar, atrophy, cerebellar-ataxia, dementia, dentatorubral-pallidoluysian-atrophy, encephalopathies, epilepsies-myoclonic, epilepsy, epilepsy-generalized, lafora-disease, myoclonus, nerve-degeneration, neurodegenerative-disorders, neuronal-ceroid-lipofuscinoses, tonic-clonic-epilepsy, trinucleotide-repeat-expansion, unverricht-lundborg-syndrome.
Among the many pathways, these few ones have gauged particular interests from scientists studying Myoclonic Epilepsies, Progressive, and have been seen in publications frequently: Aging, Amyloid Fibril Formation, Autophagy, Cell Death, Cell Division, Cognition, Dna Deamination, Excretion, Hyperphosphorylation, Immune Response, Localization, Methylation, Muscle Atrophy, Nuclear Transport, Pathogenesis, Protein Targeting, Reflex, Synaptic Transmission, Translation, Transport
Quite a number of genes have been found to play important roles in Myoclonic Epilepsies, Progressive, such as ATN1, ATXN1, ATXN3, CSF2, CSTB, EPM2A, FMN1, FOXC2, GBA, HTT, LAMC2, MUL1, NHLRC1, PPP1R3C, SCARB2, TRIM50. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.